Kin-Kee Pun, MD; Cheuk-Kit Wong, MD; Elaine Yee-Ling Tsui, MD; Sidney Chi-Fai Tam, MD; Annie Wai-Chee Kung, MD; Christina Chun-Lun Wang, MD
The Sjögren syndrome is complicated by distal renal tubular acidosis in about 40% of the patients (1). Three patients presented with hypokalemic paralysis as a result of renal tubular acidosis that occurred 2 to 7 years before xerostomia or xerophthalmia. Hypokalemic paralysis occurs more frequently in Orientals as a result of thyrotoxicosis (2, 3), hyperaldosteronism (4), barium poisoning (5), and as a side effect of glossypol, a fertility regulating agent used in China (6).
A 34-year-old southern Chinese woman was hospitalized because of sudden onset of quadriparesis caused by exertion followed by a heavy meal. On admission, there was
Pun K, Wong C, Tsui EY, Tam SC, Kung AW, Wang CC. Hypokalemic Periodic Paralysis due to the Sjögren Syndrome in Chinese Patients. Ann Intern Med. 1989;110:405–406. doi: 10.7326/0003-4819-110-5-405
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Published: Ann Intern Med. 1989;110(5):405-406.
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